In psychopharmacology, there are several beliefs that clinicians have and which seem plausible based on reasoning from pharmacological effects but turn out to not be supported by the empirical evidence available.
Simple and Practical Mental Health will subject such beliefs to careful, thorough examination. And if the belief is confirmed to be a myth, we will add it to this page.
I think it is very important to subject our beliefs to rigorous inquiry. I will refer to this field as psychopharmythology and will call such myths psychopharmyths.
What beliefs in psychopharmacology do you think are myths and belong on this page? Post your comment at the bottom of this page under “Leave a Reply”.
Reports are already ready that evaluate the following as psychopharmyths or facts:
Does antidepressant-induced sexual dysfunction tend to resolve spontaneously over time in most patients?
Does weight gain with second-generation (atypical) antipsychotics plateau after the first few months?
Are these psychopharmyths?
We will post our reviews and answer these questions soon. In the meanwhile, what do you think? Please post your comments under “Leave a Reply” at the bottom of this page.
– Can anticholinergics can be used to treat antipsychotic-induced tardive dyskinesia?
– Is topiramate the most effective treatment for antipsychotic-induced weight gain?
– Is bupropion more likely to cause seizures than other antidepressants like SSRIs?
– Is bupropion not appropriate for major depressive disorder with anxious features?
– Is bupropion less likely to worsen the course of bipolar disorder than other antidepressants?
– Do beta-blockers cause depression?
– Does nortriptyline really have a therapeutic window?
– Are TCAs really more effective than SSRIs?
– Is vilazodone less likely to cause sexual dysfunction than the SSRIs?
– Are second-generation (“atypical”) antipsychotics less likely to cause tardive dyskinesia than first-generation antipsychotics?
– Is duloxetine more likely than other antidepressants to cause liver damage?
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